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- Apr 26, 2011
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For over 40 years, the myth of the primary cause of heart disease is that cholesterol "causes" plaque build up in the arteries, leading to obstruction of blood flow, and subsequent morbidity and mortality.
Drug companies profit from USD25 billion dollars in statin drug sales, annually.
While it is true that oxidized low-density lipoprotein is found within the atheromatous plaque found in damaged arteries, it is only an effect of heart disease. The underlying damage to the lining of the artery, which could be infectious, chemical, stress and/or nutritionally-related, comes before the immune response that results in plaque buildup there. Blaming LDL cholesterol for causing heart disease, is like blaming the scab for the injury that caused it to form !
Statin drugs damage the muscles and nerves in the body -- a dose as low as 5 mg a day can kill a human !
Published in the Archives of Internal Medicine, statin drugs increase the risk of diabetes by 48%. This adds to growing clinical evidence that statin drugs are fundamentally diabetogenic.
Statin drugs contribute to insulin resistance, elevated blood sugar, and full-blown diabetes, are not only diabetogenic but cardiotoxic, as well.
Cardiotoxicity is characteristic of statin drugs. Because the most well-known adverse effect of statin drugs is their muscle-damaging (myotoxic) properties, it is commonsense to deduce that statin drugs are toxic to the heart muscle and actually weaken the heart muscle.
What is the most important nutrient widely recognized to benefit cardiovascular health? Coenzyme Q10 would be the correct answer.
And statin drugs suppress the production (via mevalonate pathway inhibition) of this indispensable factor in mitochondrial ATP production. The heart muscle is so ATP-dependent that each cardiac muscle cell has as many as 200 times higher levels of mitochondria than skeletal muscle cells. It is, after all, the muscle that never stops working.
There are a wide range of nutritional deficiencies statin drugs induce, including selenium, zinc, and vitamin E deficiency -- all of which may profoundly harm cardiovascular function.
Drug companies profit from USD25 billion dollars in statin drug sales, annually.
While it is true that oxidized low-density lipoprotein is found within the atheromatous plaque found in damaged arteries, it is only an effect of heart disease. The underlying damage to the lining of the artery, which could be infectious, chemical, stress and/or nutritionally-related, comes before the immune response that results in plaque buildup there. Blaming LDL cholesterol for causing heart disease, is like blaming the scab for the injury that caused it to form !
Statin drugs damage the muscles and nerves in the body -- a dose as low as 5 mg a day can kill a human !
Published in the Archives of Internal Medicine, statin drugs increase the risk of diabetes by 48%. This adds to growing clinical evidence that statin drugs are fundamentally diabetogenic.
Statin drugs contribute to insulin resistance, elevated blood sugar, and full-blown diabetes, are not only diabetogenic but cardiotoxic, as well.
Cardiotoxicity is characteristic of statin drugs. Because the most well-known adverse effect of statin drugs is their muscle-damaging (myotoxic) properties, it is commonsense to deduce that statin drugs are toxic to the heart muscle and actually weaken the heart muscle.
What is the most important nutrient widely recognized to benefit cardiovascular health? Coenzyme Q10 would be the correct answer.
And statin drugs suppress the production (via mevalonate pathway inhibition) of this indispensable factor in mitochondrial ATP production. The heart muscle is so ATP-dependent that each cardiac muscle cell has as many as 200 times higher levels of mitochondria than skeletal muscle cells. It is, after all, the muscle that never stops working.
There are a wide range of nutritional deficiencies statin drugs induce, including selenium, zinc, and vitamin E deficiency -- all of which may profoundly harm cardiovascular function.
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